AARS plays critical roles in maintaining cell survival and tumor progression by catalyzing the binding of specific amino acids to their respective tRNAs and activating protein translation and synthesis.[11] Our attention was focused on the top‐scoring protein, LARS1 (Figure S2A,B, Supporting Information), which is responsible for attaching leucine to its cognate tRNAs and serves as a leucine sensor to trigger mTORC1 activation.[12] Knockdown or inhibition of LARS1 can effectively suppress tumor progression.[13] Therefore, targeting LARS1 might be helpful in the treatment of OC. The gene discussed is LARS1; the disease is neoplasm.