In addition, miR-326, miR-155, and miR-23b have been demonstrated to participate in the regulation of BD through multiple signaling pathways, such as protein kinase B/mammalian target of rapamycin (Akt/mToR), Notch, and nuclear factor kappa B (NF-κB) (Gu et al. 2023). This evidence concerns the gene AKT1 and Behcet disease.