In PCa, it has been found that sEV-packaged miR-95 from TAMs is taken up by malignant cells, where it interacts with the oncogenic transcription factor JunB and favors cancer proliferation, EMT, and invasion; in accordance with these data, increased miR-95 expression in PCa tissues correlates with worse clinicopathological features [61]. Here, JUNB is linked to posterior cortical atrophy.