Moreover, the treatment with TGFβ-1 for 24 h in control and IPF fibroblasts from 10 different patients showed a homogenous and stable increase in the protein levels of COL1, but an inconsistent reaction in the case of α-SMA (Fig. 2D, Table 1). The gene discussed is TGFB1; the disease is idiopathic pulmonary fibrosis.