The frequency of new bone remodeling sites decreases, and the bone remodeling gap decreases, leading to a rapid, severe or even continuous decline in serum calcium, phosphorus and magnesium, manifesting electrolyte disorders such as secondary hypocalcemia, hypophosphatemia and hypomagnesemia.[15] Secondary hypocalcemia is associated with a secondary elevation of PTH, and hypophosphatemia is more common in postoperative patients with decreased bone resorption and increased bone formation and normal urinary phosphorus excretion. This evidence concerns the gene PTH and hypophosphatemia.