AGT and dilated cardiomyopathy: We found that during early MI, migratory myofibroblasts (IFS 2), matrifibrocytes (IFS 3) together with other proinflammatory states (IFS 6 and 7) were prioritized, in contrast to AngII and late MI which showed convergence of their profibrotic disease signatures, suggesting that independent of the initial type of tissue damage a conserved fibroblast response is evoked over time in line with reported findings on convergence of transcriptomic profiles of end-stage ischemic and dilated cardiomyopathy [75, 88].