The increased expression of endothelial adhesion molecules such as β2-integrins (CD11/CD18), ICAM-1, and p-selectin might play a role in the recruitment of leukocytes as well as platelets in the peripheral vessels following ischemic stroke [18] and potentially might lead to cell-mediated dysfunction of endothelial cells. The gene discussed is ICAM1; the disease is ischemic stroke.