After IR, MG are abnormally activated, triggering the secretion of an array of proinflammatory cytokines such as monocyte chemoattractant protein‐1 (MCP‐1), TNF‐α, ILs (IL‐1β, IL‐18 and IL‐1а), and granulocyte‐macrophage colony‐stimulating factor (GM‐CSF), and dampening the release of anti‐inflammatory cytokines such as CD86, CD32, CD206, and IL‐10, thereby amplifying the neuroinflammatory response. This evidence concerns the gene IL1B and myasthenia gravis.