JAK1 and fibrodysplasia ossificans progressiva: Results in this work show that STAT pathway genes are upregulated in cycling MSCs after muscle or tendon injury, especially targets of Stat3 and Stat1 known to play a role in cell proliferation among other processes.[51] Interestingly, inhibition of JAK1/2 could prevent neurogenic HO.[52] We noted that both traumatic HO and FOP share some common features in pathology, i.e., injury‐induced inflammatory response will promote or accelerate heterotopic endochondral bone formation in the soft tissues.