In 2003, during SARS coronavirus (SARS-CoV) pandemic, several research investigations which looked into the causes of pancreatic lesions and glucose intolerance in SARS patients, demonstrated that the presence of angiotensin-converting enzyme 2 (ACE2) in the exocrine and endocrine tissues of the pancreas indicated that SARS-CoV could destroy islets and lead to acute insulin-dependent diabetes mellitus [5–7]. The gene discussed is ACE2; the disease is Glucose intolerance.