Podocyte, a type of glomerular visceral epithelial cell, play a critical role in filtration and are involved in glomerulopathies induced by diabetic nephropathy; in H2O2-injured podocytes, Mor reduced apoptosis by downregulating Bax and decreasing the Bax/Bcl-2 ratio. Here, BAX is linked to lipoprotein glomerulopathy.