Although this last result may seem contradictory, it is known in the literature that hyperthyroidism, a condition comparable to the supraphysiologic T3 dose used in our experimental model, is associated with the activation of proteins related to the inflammatory response (109) through the release of pro-inflammatory cytokines into the bloodstream, including IL-6 (110, 111), which in turn promotes lipid peroxidation and free radical formation. Here, IL6 is linked to hyperthyroidism.