A similar situation was observed in the PQ comparison group: two DEPs (PLCB1, and NFATC2) were enriched in the noncanonical Wnt pathway and two DEPs (SERPINF1 and SMAD4) were enriched in the canonical pathway, suggesting that the activation of the noncanonical Wnt pathway is not less effective than that of the canonical pathway in both types of lung fibrosis. This evidence concerns the gene NFATC2 and pulmonary fibrosis.