However, RNA sequencing, immunoblotting, and immunostaining analyses revealed comparable transcription and translation levels among the tinnitus, control, and nontinnitus groups, suggesting that the hyperexcitability associated with tinnitus is likely mediated by abnormal KCNQ2 and KCNQ3 functioning rather than aberrant protein expression (Fig. S4). Here, KCNQ2 is linked to Tinnitus.