Interestingly, in the 1990s, during the early stages of intense molecular research on the causes of AD, the dispute over whether amyloid (plaques) or tau (tangles) is responsible for disease development divided the AD community into two camps, “Baptists” and “Tauists,” until they were ‘pathomechanistically’ united in the flow of the amyloid cascade of events (Lewis et al., 2001; Götz et al., 2001; Lee, 2001; Mudher and Lovestone, 2002). The gene discussed is MAPT; the disease is Alzheimer disease.