Moreover, Galectin-10 released by eosinophils during EETosis has been found to correlate with the degree of EETosis in BA lung tissue and nasal mucosal tissue in chronic rhinosinusitis with nasal polyps, suggesting that Galectin-10 in EVs may reflect the pathogenesis of eosinophilic inflammation in asthma (Table 1). The gene discussed is CLC; the disease is nasal cavity polyp.