STING1 and cervical squamous intraepithelial neoplasia: Continuous CIN can evoke an inflammatory response via the cGAS-STING pathway as a consequence of genetic material that ends up in the cytoplasm due to rupture of micronuclei or chromatin bridges that persist (Dou et al, 2017; Flynn et al, 2021; MacKenzie et al, 2017; Sun et al, 2013).