Increased gut permeability is responsible of MT and even a low-grade endotoxemia may induce production of host response molecules such as lipopolysaccharide-binding protein (LBP) and consumption of neutralizing antibodies against lipopolysaccharide (LPS) endotoxin core antigen [antiendotoxin core antibody (EndoCab)] [23]. This evidence concerns the gene LBP and serum lipopolysaccharide activity.