TNKS2 and lymphoma: EBV latency in naïve B cells is thought to promote both cHL and extranodal TNKL through expression of viral genes including EBNA-1, EBNA-LP, latent membrane protein-1, -2A, and -2B, EBERs, and BARTs, comprising the so-called EBV latency type II gene pattern; separate sets of EBV genes and distinct latency types are associated with other lymphomas [9].