The specific mechanism was that METTL3 mediated the m6A modification of the untranslated region at the 5′ end of histone Tissue L (CTSL) mRNA, and in turn, the m6A reading protein Insulin-like Growth Factor 2 (IGF2BP2) binds to the m6A site, which enhances the stability of the CTSL mRNA, thus promoting the metastasis of cervical cancer cells. The gene discussed is CTSL; the disease is cervical carcinoma.