Although the lack of STING did not affect animal survival and despite similar levels of IFNβ mRNA, STING KO mice displayed significantly higher viral NiV-N mRNA levels in all tested organs, followed by lower expression of CXCL10 in the brain and lung in the mid phase of infection compared to WT mice, as observed in IFNAR KO control mice (Fig 1C). Here, STING1 is linked to infection.