VIM and head and neck squamous cell carcinoma: The decrease of H3K9ac is related to the activation of epithelial-mesenchymal transition (EMT) and the increased cell proliferation during the process of oral carcinogenesis, indicating the H3K9ac is involved in the progression of HNSCC and is coexpressed with the mesenchymal vimentin prior to the invasion of HNSCC [30].