Arguments for a pathophysiological role of anti-recoverin antibodies are that the patient was temporarily blind, had permanent visual impairment, amblyopia, reduced dim vision, and color vision deficiency, that anti-recoverin antibodies were repeatedly elevated in serum and cerebrospinal fluid, and that anti-recoverin antibody syndrome has been previously described as retinopathy, uveitis, and cerebellar syndrome [14]. This evidence concerns the gene RCVRN and color vision disorder.