The knockdown of SULT2B1b in vitro suppresses tumor cell growth and promotes apoptosis and cell cycle arrest in Hepa1-6 cells by increasing the pro-apoptotic factor (FAS) expression, and downregulating the anti-apoptotic factor BCL-2, cyclinB1, and MYC, promoting cancer cell death (Yang et al., 2013). This evidence concerns the gene MYC and cancer.