–↓ cellular lipid accumulation in both types of hepatic steatosis (FA and T090);–↑ cell survival/proliferation with a peak effect at 1 μg/mL;–↓ Rubicon (protein inhibiting late-stage autophagy) –↑ p62/SQSTM1 (autophagy marker);–↓ the cellular FA synthesis pathway stimulated by the LXR agonist (nuclear receptor transcription factor involved in lipid synthesis);. This evidence concerns the gene SQSTM1 and Hepatic steatosis.