SOD1 and epilepsy: In this context, the concentration of H2O2 in the brain increases in the early stages of epilepsy due to the action of SOD, which is responsible for the dismutation of superoxide radicals (O2•−) into H2O2, after which the concentration of this oxidant compound decreases due to the action of other antioxidant enzymes [28,34,35,36,37].