Oxidative stress was established as a significant contributing factor in the etiology of ADHD [36,37], and the nuclear receptor Nr2f1 of the inflammatory pathway plays important functions in specifying diverse neuron subtypes throughout the patterning of the neocortical motor and somatosensory cortex, in addition to the regulation of the longitudinal hippocampal growth during development [32]. The gene discussed is NR2F1; the disease is attention deficit-hyperactivity disorder.