Findings support the view that PPARγ supports PCa cell growth by increasing the machinery for lipid synthesis, including cutaneous fatty acid-binding protein (C-FABP) [54], fatty acid synthase (FASN), ATP citrate lyase (ACLY), and mitochondrial biosynthesis [55], bringing to attention that NRs regulate PCa metabolism. The gene discussed is ACLY; the disease is posterior cortical atrophy.