ARID1A behaves as a tumour suppressor gene in OCCC, with both alleles presumed to be affected by concomitant variants, specifically mutations (deletions and insertions as well as nonsense mutations, all leading to premature STOP codons that would truncate the normal protein) and loss of heterozygosity, or the presence of presumably biallelic mutations [14,76]. This evidence concerns the gene ARID1A and neoplasm.