In addition, responders have an activation vs. non-responders of the “Neuroprotective Role of THOP1 in Alzheimer’s Disease”, involved in the neuroprotective response to amyloid beta toxicity [50], and the “Role of MAPK Signalling in Inhibiting the Pathogenesis of Influenza”, important in influenza-virus infections and involved in several immune-related processes such as cellular proliferation, differentiation, inflammation, and death [51]. The gene discussed is THOP1; the disease is Alzheimer disease.