For example, members of the TNF superfamilies (TNFSF, mainly 9, 10, and 15) are molecular mechanisms involved in the “Wound Healing Signalling Pathway”, the “Systemic Lupus Erythematosus In B Cell Signalling Pathway”, the “Pathogen Induced Cytokine Storm Signalling Pathway” and the “Multiple Sclerosis Signalling Pathway”, all activated in these comparisons. The gene discussed is TNF; the disease is multiple sclerosis.