Since the increased IL-2 signalling in CD8+ T cells during viral infections prioritises robust immune response against production of long-lived memory cells, this explanation might also explain why rs61839660 near IL2RA strongly co-localised between the risk of type 1 diabetes and the eQTL of IL2RA in effector memory T cells and CD8+ naive/central memory T cells. The gene discussed is IL2; the disease is viral infectious disease.