Systemic reparixin treatment largely attenuated the tumor‐promoting effect of SKAP1 (Figure 5D,E) and completely abolished SKAP1‐associated infiltration of neutrophils in HCT116 tumors (Figure 5F), suggesting that SKAP1 promotes neutrophil infiltration into colon tumors via CXCL8. The gene discussed is CXCL8; the disease is colonic neoplasm.