Systemic reparixin treatment largely attenuated the tumor‐promoting effect of SKAP1 (Figure 5D,E) and completely abolished SKAP1‐associated infiltration of neutrophils in HCT116 tumors (Figure 5F), suggesting that SKAP1 promotes neutrophil infiltration into colon tumors via CXCL8. This evidence concerns the gene SKAP1 and neoplasm.