SKAP1 and neoplasm: This may be due to increased infiltration of classical neutrophils, which can transform to CD14+ PMN‐MDSCs locally in the tumor microenvironment.[21] PMN‐MDSCs can also create NETs[44] and be eliminated by anti‐Ly6G antibody injection, therefore, we cannot discount the potential involvement of PMN‐MDSCs in SKAP1‐induced tumor promotion and NET formation.