The increase in expression of crucial neurotrophic and neuroprotective genes, such as Bdnf, Inhibin beta A, and Npas4 [1, 6, 22, 40], contributes to the ability of FP802 to counteract ALS disease progression, underscoring its therapeutic potential in neurodegenerative conditions. This evidence concerns the gene BDNF and amyotrophic lateral sclerosis.