Moreover, we identified a trend towards higher CD3 + T cell counts among aspirated mice, which are a potential source of IL-17 (i.e., Th17 cells and monocyte/macrophage accessory cells), a proinflammatory cytokine that has been associated with pulmonary fibrosis and progressive airway obliteration [32, 33]. The gene discussed is IL17A; the disease is pulmonary fibrosis.