For this purpose, we examined pancreatic tissues from mice administered repeated 16 doses of poly(I:C) in the usual AIP induction regimen, but in this case in combination with anti-pDC Ab (120G8 Ab), anti–IFN-α/β receptor Ab (anti-IFNAR Ab), anti-CXCR3 Ab, and anti–IL-33 receptor Ab (anti-ST2 Ab) and then evaluated the expression of CCL25 in pancreatic tissues by blindly counting immunoreactive CCL25+ cells that were morphologically identified as lymphoid cells (4, 5). This evidence concerns the gene CXCR3 and autoimmune pancreatitis.