It is not purely cardio specific, since it’s also present in skeletal muscle, brain, and kidneys.4 The release of H-FABP into the bloodstream occurs as a result of myocardial injury, and it can be detected from a blood sample.4 H-FABP serum levels in healthy people are typically in the single digit ng/ml range.5-7 The microRNA miR-1 controls the expression of H-FABP and may contribute to the development of heart failure itself.8 Rapid release of H-FABP from myocytes into the systemic circulation is seen subsequent to myocardial damage. This evidence concerns the gene FABP3 and heart failure.