SELE and malaria: During the erythrocytic phase of malaria pathogenesis, P. falciparum‐erythrocytes express PfEMP‐1 surface antigen which facilitates the adherence of infected erythrocytes to endothelial surfaces by binding to endothelial cell adhesion molecules such as vascular cell adhesion molecule‐1, intercellular adhesion molecule‐1, P‐selectin and E‐selectin.8