FOXO1 and myocardial infarction: found that overexpression of circRNA FEACR could inhibit MI and improve cardiac function by directly binding to nicotinamide phosphoribosyltransferase (NAMPT), improving NAMPT protein stability, enhancing SIRT1 expression, and promoting forkhead box protein O1 (FOXO1) transcriptional activity, thereby upregulating the transcription of the ferroptosis inhibitor Fth1.