The stages of bone marrow B‐cell development where WAS patient shows failure to expand positively selected cells are stages with low antiapoptotic protein iBcl‐2, opening questions of WAS protein role in tuning antiapoptotic signaling in developing B cells since increased apoptosis was described in human WAS patients’ peripheral lymphocytes [31]. The gene discussed is WAS; the disease is Wiskott-Aldrich syndrome.