However, in a study by Qin et al. (2015), administration of Ang-(1–7) reduced EGFR expression and activity in the kidney in rats with experimental glomerulonephritis supporting the notion that Ang-(1–7) is an inhibitor of EGFR and that this might represent a potential mechanism by which it exerts its beneficial renal effects. Here, EGFR is linked to glomerulonephritis.