MASLD was diagnosed following an unremarkable comprehensive evaluation that ruled out hepatitis B and C infections, documented normal autoimmune hepatitis profile (anti-smooth muscle antibody, anti-nuclear antibody, liver kidney microsome antibodies, mitochondrial M2 antibody, antineutrophil cytoplasmic antibody, soluble liver antigen antibody and IgG of 1201 mg/dL), A1AT phenotype was M1M2 (level 123 mg/dL), Wilson’s disease was unlikely due to normal ceruloplasmin (31 mg/dL), and screening for celiac disease was negative (TTG IgA 0, total IgA 120 mg/dL). This evidence concerns the gene CD79A and Wilson disease.