Therefore, to define whether the outgrowth phenotype of mycobacteria in IFNγ-expressing macrophages is due to a host-mediated impact of IFNγ or to a direct effect of IFNγ per se, we supplemented antibodies against CD119 to intercept the host IFNγ signaling (Fig. S3) following infection of control or IFNγ-expressing macrophages with M. bovis BCG and then analyzed the intracellular growth of mycobacteria. This evidence concerns the gene IFNGR1 and infection.