SLC1A2 and type 2 diabetes mellitus: Overexpression of GLT‐1 in the hippocampal astrocytes significantly attenuated the hyperexcitability of hippocampal glutamatergic neurons, reduced oxidative stress and neuronal apoptosis, improved LTP and cognitive function in T2DM mice suffering anesthesia/surgery, which suggested that the excitotoxic injuries of glutamatergic neurons in hippocampus induced by reduced expression of GLT‐1 in astrocytes may trigger the cognitive dysfunction in T2DM mice after isoflurane anesthesia/surgery.