IFNGR1 and granular corneal dystrophy type II: Our results identified a cluster of CXCL9/10-producing dFBs/pAds in ACD, and showed that targeted deletion of Ifngr1 in PDGFRA+ dFB not only reduced DNFB-induced Cxcl9/10 production but also reduced trafficking of CD8+ T cells to affected skin sites.