TGM2 and cranioectodermal dysplasia: Doğan et al. reported that CeD patients with seropositivity with anti-endomysial antibodies (anti-EmA) and anti-tissue transglutaminase type two antibodies (anti-TG2) had a thinner subfoveal choroidal thickness than non-seropositive CeD patients, although their findings were statistically insignificant and the authors could not prove the presence of autoantibodies in the choroidal microcirculation [32].