Galactose-deficient IgA1 (Gd-IgA1) and related IgA/IgG containing circulating immune complexes (CIC) were identified as the key drivers in the pathogenesis of secondary IgAN similar to primary IgAN.8 Our patient had elevated IgA levels and the pathogenesis of IgAN in pSS might be attributed to the deposition of IgA-containing CIC. This evidence concerns the gene IGHA1 and peeling skin syndrome.