They also suggested that miR-195 could suppress proliferative vascular disease through NF-kB pathways and intracellular signalling cascades such as p38.47 Serum miR-195 level is a potential indicator for thrombosis developing after acute myocardial infarction and orthopaedic surgeries.48,49 Interestingly, in our study, vascular-Behçet patients had higher miR-195 expression levels than BD patients without vascular involvement. The gene discussed is NFKB1; the disease is myocardial infarction.