NFKB1 and neoplasm: On the other hand, the pathways enriched in C2 included TNF‐α signalling via NF‐κB, inflammatory response, hypoxia, EMT, mTORC1 signalling, and IL6‐JAK‐STAT3 signalling, which were associated with tumour metastasis and growth (Figure 5E).69, 70, 71, 72