The CDK family members, such as CDK1 and CDK6, are associated with active cell cycle and play crucial roles in promoting PASMC proliferation.[33] Therefore, inhibition of CDKs may be beneficial to attenuate PAH.[33] In the present study, we further found that increasing miR‐30d significantly inhibited PDGF‐induced proliferation and migration of hPASMC in vitro. Here, CDK1 is linked to pulmonary arterial hypertension.