In vivo tracking experiment showed that when BM-MSC were transplanted into a mouse model of 60 min of transient middle cerebral artery occlusion (tMCAO), these cells quickly penetrated into the lungs and disappeared within 24 h, leaving migrasomes enriched in the antimicrobial peptide dermcidin (DCD) to enhance macrophage LC3-associated phagocytosis (LAP) and improve bacterial clearance (Li et al., 2023a). The gene discussed is DCD; the disease is cerebritis.